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Dallas McCarver, IFBB professional bodybuilder, died after an unwitnessed cardiac event contributed to by of combination of coronary atherosclerosis (a buildup of plaque in the arteries) and an enlarged left ventricle (a thickening of the heart muscle that causes it to work harder). McCarver had a family history of cardiovascular disease including both hypertension and atherosclerosis.
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Dallas was found on the floor in his living room with food scattered around his body shortly after midnight on August 22nd, 2017. Paramedics were called and he arrived at the hospital in full cardiac arrest. He was pronounced dead at 1:03 A.M. An autopsy was performed the following day.
Noted at the time of autopsy was an enlarged liver and kidneys, nephrosclerosis (a hardened kidney), heavy lungs, and a papillary thyroid carcinoma. Prior medical history includes cholesterol issues (high LDL/ low HDL), elevated aminotransferase levels, a chronic cough and shortness of breath, and childhood asthma.
His testosterone levels were very high. Trenbolone metabolites were also present. Screening for additional steroids was negative, although he tested positive for caffeine and marijuana metabolites (neither of which are mentioned as contributing factors in his death). No other recreational drugs or narcotics were present.
Also noted (but neither tested for, nor listed as a contributing factor in Dallas’ death) was a prior history of hGH and insulin use.
7cf8887de122b6e379b6effa84a152b0.jpg
Dallas was found on the floor in his living room with food scattered around his body shortly after midnight on August 22nd, 2017. Paramedics were called and he arrived at the hospital in full cardiac arrest. He was pronounced dead at 1:03 A.M. An autopsy was performed the following day.
Noted at the time of autopsy was an enlarged liver and kidneys, nephrosclerosis (a hardened kidney), heavy lungs, and a papillary thyroid carcinoma. Prior medical history includes cholesterol issues (high LDL/ low HDL), elevated aminotransferase levels, a chronic cough and shortness of breath, and childhood asthma.
His testosterone levels were very high. Trenbolone metabolites were also present. Screening for additional steroids was negative, although he tested positive for caffeine and marijuana metabolites (neither of which are mentioned as contributing factors in his death). No other recreational drugs or narcotics were present.
Also noted (but neither tested for, nor listed as a contributing factor in Dallas’ death) was a prior history of hGH and insulin use.
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