what does everyone think of this...

[Governor]

TweetSome thing ive never heard of for joints and stuff. If it works my knees could really use it.


The difference between the activity of EFA's and cetyl myristoleate is that the quantity required and the period of time over which EFA's are taken are markedly longer. Cetyl myristoleate is taken in a one month course of about 13 grams, while EFA's must be taken over extended periods, sometimes many years, and intake varies widely from hundreds to thousands of grams. Cetyl myristoleate seems to have properties in common with EFA's, but it acts faster and lasts longer.

Simplified theoretical explanation of CMO action

CMO serves as a surfactant and not only lubricates the involved joints, but also lubricates the entire body, making muscles glide more smoothly over other muscles, bursas, and bones and at the same time softens these tissues making them more pliable. Next, it functions as an immune system modulator. This is the reason it has been found to be so effective in treating auto-immune diseases such as rheumatoid arthritis, systemic lupus erythematosus, and multiple sclerosis. And finally, it functions like a fatty acid in that it mediates inflammatory processes. When cooled, cetyl myristoleate is a waxy substance and, at room temperature, has a buttery consistency.





Cetyl myristoleate
Potential treatment plan for
osteoarthritis and rheumatoid arthritis


Cetyl myristoleate, an oil, is the hexadecyl ester of the unsaturated fatty acid cis-9-tetradecenoic acid. The common name for the acid is myristoleic acid. Myristoleic acid is found commonly in fish oils, whale oils, dairy butter, and kombo butter. The chemical formula for cetyl myristoleate is (Z)-ROCO(CH2)7CH =CH(CH2)3CH3. Cetyl myristoleate was unrecorded in chemical literature until Diehl's discovery was reported. The current Merck Index of Chemicals does not list cetyl myristoleate. A search of Chemical Abstracts lists Diehl's method of extracting cetyl myristoleate from mice but contains no reference to cetyl myristoleate prior to his 1977 patent.

The exact mechanism of cetyl myristoleate's physiologic activity is unclear. As a fatty acid ester, it appears to have the same characteristics as the essential fatty acids, linoleic and alpha linolenic acids, except stronger and longer lasting. These fatty acids are referred to as "essential fatty acids" because the human body cannot make them and we must ingest them in our diets. These EFA's truly are essential to normal cell structure and body function and function as components of nerve cells, cell membranes, and hormone-like substances known as prostaglandins. Many of the beneficial effects of a diet rich in plant foods is a result of the low levels of saturated fat and the relatively higher levels of EFA's. While a diet high in saturated fat has been linked to many chronic diseases, a diet low in saturated fat but high in EFA's prevents these very same diseases. The use of EFA's over an extended period of time has been shown to decrease the pain, inflammation, and limitation of motion of arthritis.

The difference between the activity of EFA's and cetyl myristoleate is that the quantity required and the period of time over which EFA's are taken are markedly longer. Cetyl myristoleate is taken in a one month course of about 13 grams, while EFA's must be taken over extended periods, sometimes many years, and intake varies widely from hundreds to thousands of grams. Cetyl myristoleate seems to have properties in common with EFA's, but it acts faster and lasts longer.

In chronic inflammatory processes, the supply of EFA's is depleted. Cetyl myristoleate appears to have the ability to correct the imbalance created by chronic inflammation. Like EFA's, maybe cetyl myristoleate turns off the fires of chronic inflammation by serving as a mediator of prostaglandin formation and metabolism.

Nearly all gastrointestinally absorbed products initially pass through the liver, and most are extracted or modified before passage into systemic circulation. Since all fatty acids enter systemic circulation through the liver, an oil like cetyl myristoleate would begin its systemic circulation from the liver also. It is speculated that cetyl myristoleate stimulates the production of immunoglobulins and series 1 and 3 prostaglandins, which could be one explanation for why cetyl myristoleate has such potent effect in auto-immune and inflammatory conditions.

https://www.nchc2000.org/articles/cmo.html

[Governor]

Tweetbtw...I was looking around at bodybuilding.com and fount this. Never heard of it before so i figured id ask you guys...

[paperbolix]

Tweetwhat i think is, it's technical and complicated and pretty boring; but thax for the info

[trapattack369]

Tweetgo get a script for
indomethacin, take care of all your joint probs