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    1. #1
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      hey guys what's up I have a quick question for you because I have had a tremendous trouble finding an answer to this I've looked everywhere and I can't find one maybe one of you have tried the stuff. I have 100 tabs of what do they call it methotry something it's actually oral tren. it is . 25 MCGS a tab.
      one article I read said is the strongest steroid on Earth by far and another article I read said it sucks and the side effects are so bad you won't be able to finish the cycle and even if you do tren alone a is better anyway.
      if I knew it was worth it I would try to stuff I'd keep it at 25 or 5 the most. I want to try this stuff but I haven't heard of real positive thing about it so if anybody has any experience with this stuff I mean they sell it it's out there so somebody must be using it

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    2. #2
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      Default Re: question

      Quote Originally Posted by pgc648 View Post
      hey guys what's up I have a quick question for you because I have had a tremendous trouble finding an answer to this I've looked everywhere and I can't find one maybe one of you have tried the stuff. I have 100 tabs of what do they call it methotry something it's actually oral tren. it is . 25 MCGS a tab.
      one article I read said is the strongest steroid on Earth by far and another article I read said it sucks and the side effects are so bad you won't be able to finish the cycle and even if you do tren alone a is better anyway.
      if I knew it was worth it I would try to stuff I'd keep it at 25 or 5 the most. I want to try this stuff but I haven't heard of real positive thing about it so if anybody has any experience with this stuff I mean they sell it it's out there so somebody must be using it

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      it's pretty early I just posted this but I did not expect the response I've asked this question on a couple of boards and got zero feedback I think people either too scared to try it I think that's what it is people are too scared to try it so I might just go ahead and try it fuck it what's the worst that could happen it doesn't work right my liver is not going to fall out I'll take 25 micrograms just see what it does

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    3. #3
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      Default Re: question

      oral I don't think you will get much out of it at that dose

    4. #4
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      Default Re: question

      Quote Originally Posted by dirtwarrior View Post
      oral I don't think you will get much out of it at that dose
      from what I read you're right some people say it does very little with the methylated part put into the tren structure. but it's not because of the dose the normal dose is 5 mcgs a day from what I read when people first started trying it to see if it would work they went all the way up to 5 mg and those people started getting really high liver enzymes elevations so they had to actually keep cutting it back all the way to micrograms so if you look it up the recommended dose is 0.5 micrograms up to the most 1 mg but I don't think I'm going to use it cuz I have yet to hear one positive thing about it

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    5. #5
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      Default Re: question

      It has to be 17AA/methylated to be orally active. As such and probably the reason it is not mainstream is i would guess it is brutally toxic to the liver.

      If you take it i would do no more than 2 weeks on and preferably 2 weeks off. Also take liver protectants. the3y help but are not magic.
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    6. #6
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      Default Re: question

      Quote Originally Posted by jipped genes View Post
      It has to be 17AA/methylated to be orally active. As such and probably the reason it is not mainstream is i would guess it is brutally toxic to the liver.

      If you take it i would do no more than 2 weeks on and preferably 2 weeks off. Also take liver protectants. the3y help but are not magic.
      yo man thanks for the input you're really the first one that gave me any decent input on it. I can't write a lot because I'm a new member and I can really only write a couple paragraphs so I'll try to make this quick. for 20 years I've been searching for one single case in the American journal of Medicine the Harvard journal medicine the AMA monthly periodical I mean real publications that doctors read to try to find one single case of somebody going into liver failure or cirrhosis from using double A calculated oral steroids and I have yet to find a single case if you know of one I would be very grateful if you forwarded it because I'm telling you it's been around 20 years now and I can't find one. I have a theory of why they were saying it it's toxic to the liver I just don't think it's true and it's not based on any real science it's based on assumptions but I can't write a lot like I said so just thank you for your feedback and if I do use it I'll keep everybody informed

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    7. #7
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      Default Re: question

      so from what i have read and heard it is supposed to be super nasty toxic. i have heard palumbo and some others talk about it and not in a good way. 1-2 weeks tops and that is supposed to be still risky. i cant speak from personal experience but you dont seem to have issues with compounds that most do. so you may be good with it. from what i gather palumbo and others say why run the risk of it messing you up and just go with regular tren of some ester instead of stressing your liver. be curious to see how you do and feel on it.
      i will say on a side note everyone says the same thing about dnp but if done correctly i personally feel like it can be safer than clen for some people. then again that's just my opinion on it
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    8. #8
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      Quote Originally Posted by guns01 View Post
      so from what i have read and heard it is supposed to be super nasty toxic. i have heard palumbo and some others talk about it and not in a good way. 1-2 weeks tops and that is supposed to be still risky. i cant speak from personal experience but you dont seem to have issues with compounds that most do. so you may be good with it. from what i gather palumbo and others say why run the risk of it messing you up and just go with regular tren of some ester instead of stressing your liver. be curious to see how you do and feel on it.
      i will say on a side note everyone says the same thing about dnp but if done correctly i personally feel like it can be safer than clen for some people. then again that's just my opinion on it
      Thanks Guns.
      Yes, I've read pretty much the same that just using TREN A is the better option.
      We'll see, maybe I'll throw it in for 2 weeks right at a sticking point.

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    9. #9
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      Default Re: question

      wow

    10. #10
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      Default Re: question

      Quote Originally Posted by guns01 View Post
      so from what i have read and heard it is supposed to be super nasty toxic. i have heard palumbo and some others talk about it and not in a good way. 1-2 weeks tops and that is supposed to be still risky. i cant speak from personal experience but you dont seem to have issues with compounds that most do. so you may be good with it. from what i gather palumbo and others say why run the risk of it messing you up and just go with regular tren of some ester instead of stressing your liver. be curious to see how you do and feel on it.
      i will say on a side note everyone says the same thing about dnp but if done correctly i personally feel like it can be safer than clen for some people. then again that's just my opinion on it
      thanks guns you're always good for a good input from a question. yeah it is very little to be found on the internet about this stuff I found one article saying it's the most anabolic androgenic substance on Earth that it's like six times more potent than injectable trend which I don't believe. then I've heard things that say the methylated compound used to make it orally available to the body wreck some of trends positive aspects so actually the injectable will give you more results than you are why the oral is 10 times more toxic. at this point I ended up ordering four bottles of tren a so 40ccs.
      I'm probably just going to toss the oral tren.

      one thing though I didn't read that I heard one person allude to and everything I read which is a lot nobody complained about bad gains from it that it didn't work what they complained about was the side effects or the liver toxicity compared to injectable trend but nobody really commented on if it was better than injectable training if it's worth taking if you're willing to take the risk I couldn't find anything like that. that's the only thing that makes me want to try it so. I like you said guns I handle antibiotics very well I barely get and p

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    11. #11
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      so this is what i dug up from the anabolics ref guide

      Metribolone (methyltrienoloneJ
      Androgenic 6,000-7,000
      Anabolic 12,000-30,000
      ~ Standard Methyltestosterone (oral)
      Chemical Names 17alpha-methyl-17betahydroxyestra-4,9,11-triene-3-one 17alpha-methyl-trenbolone
      Estrogenic Activity none
      Progestational Activity no data available
      Description:
      Methyltrienolone is one of the strongest oral anabolic steroids ever produced. This agent is a derivative of trenbolone (trienolone), which has been c-17 alpha alkylated to allow for oral administration.This modification has created a AAS that is significantly stronger than its non-methylated cousin. Its potency has been measured to be anywhere from 120-300 times greater than that of methyltestosterone, with greater dissociation between anabolic and androgenic effects.625 626 Milligram for milligram methyltrienolone is a more active AAS than any agent sold on the commercial market, requiring doses as little as .5-1 milligram per day to notice a strong anabolic effect. Its potency is only matched by its relative toxicity, however, which has limited its modern use to that of laboratory research only.
      History:
      Methyltrienolone was first described in 1965.627 It was immediately identified as an extremely potent anabolic agent, far more potent than the commercially available agents of the time. In spite of its high relative activity, however, methyltrienolone has seen very limited use in humans. It was used clinically during the late 1960's and early '70's, most notably in the treatment of advanced breast cancer. Here, its exceedingly strong anabolic/androgenic action helps the drug counter the local effects of endogenous estrogens, lending it some efficacy for slowing or even regressing tumor growth. Such application was not long lived, however, as more realistic evaluations of the drug's toxicity soon led to its
      , abandonment in human medicine.
      By the mid-1970's, methyltrienolone was becoming an accepted standard in non-human research studies, particularly those pertaining to the study of the androgen receptor activity. For this purpose the agent is very well suited. Its sheer potency and resistance to serum-binding proteins makes it an excellent in-vitro receptor-binding
      OH
      Methyltrienolone
      standard to compare other agents to. Being so resistant to metabolism, active methyltrienolone metabolites are also not going to greatly interfere with the results of most experiments. Body tissues can metabolize most steroids fairly easily, which means that even incubation studies can be complicated with the question of what is causing a particular effect, the AAS or one of its unidentified metabolites. This is much less of an issue with methyltrienolone. Today, methyltrienolone remains an agent of research use only.
      How Supplied:
      Methyltrienolone is not available as a commercial agent.
      Structural Characteristics:
      Methyltrienolone is a modified form of nandrolone. It differs by: 1) the addition of a methyl group at carbon 17alpha to protect the hormone during oral administration and 2) the introduction of double bonds at carbons 9 and 11, which increases its binding affinity and slows its metabolism. The resulting AAS is significantly more potent than its nandrolone base, and displays a much longer half-life and lower affinity for serum-binding
      ,proteins in comparison. Methyltrienolone chemically differs from trenbolone only by the addition of a methyl group at c-17. This alteration changes the activity of methyltrienolone considerably, however, such that this agent should not simply be considered an oral form of trenbolone.
      Side Effects (Estrogenic):
      Methyltrienolone is not aromatized by the body, and is not measurably estrogenic. It is of note, however, that methyltrienolone displays significant binding affinity for the progesterone receptor.628 The side effects associated with progesterone are similar to those of estrogen, including negative -feedback inhibition of testosterone production and enhanced rate of fat storage. Progestins
      305
      .............v •• v •• ~ ••v _, __a
      also augment the stimulatory effect of estrogens on mammary tissue growth. There appears to be a strong synergy between these two hormones here, such that gynecomastia might even occur with the help of progestins, without excessive estrogen levels. The use of an anti-estrogen, which inhibits the estrogenic component of this disorder, is often sufficient to mitigate gynecomastia caused by progestational anabolic/androgenic steroids.
      Side Effects (Androgenic):
      Although classified as an anabolic AAS,androgenic side effects are stHI common with this substance. This may include bouts of oily skin, acne, and body/facial hair growth. Anabolic/androgenic steroids may also aggravate male pattern hair loss. Women are also warned of the potential virilizing effects of anabolic/androgenic steroids. These may include a deepening of the voice, menstrual irregularities, changes in skin texture, facial hair growth, and clitoral enlargement. Additionally, the 5-alpha reductase enzyme does not metabolize methyltrienolone, so its relative androgenicity is not affected by finasteride or dutasteride.
      Side Effects (Hepatotoxicity):
      Methyltrienolone is a c17-alpha alkylated compound. This alteration protects the drug from deactivation by the liver, allowing a very high percentage of the drug entry into the bloodstream following oral administration. e17-alpha alkylated anabolic/androgenic steroids can be hepatotoxic. Prolonged or high exposure may result in liver damage. In rare instances life-threatening dysfunction may develop. It is advisable to visit a physician periodically during each cycle to monitor "liver function and overall health. Intake of c17-alpha alkylated steroids is commonly limited to 6-8 weeks, in an effort to avoid escalating liver strain.
      Methyltrienolone is an exceedingly potent oral AAS, with a very high level of resistance to hepatic metabolism. This makes methyltrienolone exceedingly liver-toxic, precluding its use as a prescription agent at this time, in any part of the world. Studies published from the University of Bonn Germany back in 1966 make this very clear.629 In fact, at this time researchers had deemed this the most liver-toxic AAS to ever be studied in humans, summing up their findings well when stating:
      JlMethyltrienolone... which is orally active as an anabolic agent in a dose less than 1.0 mg per day in normal adults, has been tested with regard to its influence on liver function. As measured by multiple parameters (BSP retention; total bilirubin; activities of transaminases, alkaline phosphates and
      306
      cholinesterase in serum; activity of proaccelerin in plasma) methyltrienolone turned out to be very active as to causing biochemical symptoms of intrahepatic cholestasis....thus methyltrienolone at present being the most 'hepatotoxic' AAS."
      The use of a liver detoxification supplement such as Liver Stabil, Liv-52, or Essentiale Forte is advised while taking any hepatotoxic anabolic/androgenic steroids.
      Side Effects (Cardiovascular):

      Anabolic/androgenic steroids can have deleterious effects on serum cholesterol. This includes a tendency to reduce HDL (good) cholesterol values and increase LDL (bad) cholesterol values, which may shift the HDL to LDL balance in a direction that favors greater risk of arteriosclerosis. The relative impact of an anabolic/androgenic AAS on serum lipids is dependant on the dose, route of administration (oral vs. injectable), type of AAS (aromatizable or non-aromatizable), and level of resistance to hepatic metabolism. Although not extensively studied in humans, the oral route, high relative potency, and non-aromatizable nature of methyltrienolone suggest that this agent is extremely prone to negatively altering lipid values and increasing atherogenic risk. Anabolic/androgenic steroids may also adversely affect blood pressure and triglycerides, reduce endothelial relaxation, and support left ventricular hypertrophy, all potentially increasing the risk of cardiovascular disease and myocardiai infarction.
      To help reduce cardiovascular strain it is advised to maintain an active cardiovascular exercise program and minimize the intake of saturated fats, cholesterol, and simple carbohydrates-at all times during active AAS administration. Supplementing with fish oils (4 grams per day) and a natural cholesterol/antioxidant formula such as Lipid Stabil or a product with comparable ingredients is also recommended.
      Side Effects (Testosterone Suppression):
      All anabolic/androgenic steroids when taken in doses! sufficient to promote muscle gain are expected to! suppress endogenous testosterone production. Withou~ the intervention of testosterone-stimulating substances! testosterone levels should return to normal within 1-1 months of drug secession. Note that prolongeo
      hypogonadotrophic ~econda~y to AAS Intervention.
      hypogonadism abuse, necess
      can itating
      develo~ mediCal I
      I
      The above side effects are not inclusive. For more detaile~ discussion ofpotentialside effects,see theSteroid Side Effec~ section ofthis book. I
      William Llewellyn's ANABDLICS, 9th ed.
      Administration (General):
      Studies have shown that taking an oral anabolic AAS with food may decrease its bioavailability.630 This is caused by the fat-soluble nature of AAS hormones, which can allow some of the drug to dissolve with undigested dietary fat, reducing its absorption from the gastrointestinal tract. For maximum utilization, methyltrienolone should be taken on an empty stomach.
      Administration (Men):
      Methyltrienolone is no longer used in clinical medicine due to an unacceptable level of hepatotoxicity.This agent is generally not recommended for physique-or performance-enhancing purposes for the same reason. Those absolutely insisting on its use need to take its level of liver toxicity very seriously. At the very least, routine blood tests should be conducted to ensure the agent is not imparting damage. Drug duration should also be very limited, preferably to 4 weeks of use or less. The relative potency of methyltrienolone is extremely high, requiring doses as little as .5 milligram per day. Its effective and tolerable range is usually considered to be .5 to 2mg per day. Dianabol-type doses of 20-30 mg daily are completely unthinkable, and should never be attempted. Again, this is an extremely toxic AAS, and all good advice would say to avoid it. Anyone of the many commercially available steroids would be much safer choices.
      Administration (Women):
      Methyltrienolone is no longer used in clinical medicine
      due to an unacceptable level of hepatotoxicity.This agent
      is not recommended for women for physique-or
      !
      performance-enhancing purposes due to its extremely
      strong toxicity and tendency to produce virilizing side
      effects.
      Availability:
      Methyltrienolone is not produced as a prescription AAS
      product in any part of the world. With the rapid expansion
      of underground AAS manufacturers, this agent has
      been released as a black market designer compound.
      Those contemplating the use of underground forms of
      methyltrienolone should consider that such agents are
      being released for human use without any government
      approval or consideration to its safety.
      307
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